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5.6.4. Metformin – In vitro and vivo

With respect to the mechanisms of action metformin in animals, vitro and vivo animal testing has failed to provide evidence of drug interactions metformin and glucagon.

5.6.5. Cholestyramine – In vitro and vivo

In vitro and vivo animal testing has shown a lack of drug interaction with metformin, cholestyramine, or either compound with thiazolidinediones lithium and carbon tetrachloride (at doses similar to those used for these trials), with the exception of combination bafilomycin.

5.6.6. Topiramate – In vitro and vivo

With respect to the mechanisms of action topiramate, in vitro and vivo animal testing has demonstrated lack of drug interaction with metformin, cholestyramine, or either compound with thiazolidinediones lithium and carbon tetrachloride (at doses similar to those used for these trials).

5.6.7. Cefaclor – In vitro and vivo

With respect to the mechanisms of action cefaclor, in vitro and vivo animal testing has demonstrated no evidence of drug interaction metformin, cholestyramine, or either compound with thiazolidinediones lithium and carbon tetrachloride (at doses similar to those used for these trials).

5.6.8. Diflunisal – In vitro and vivo

In vitro and vivo animal testing has failed to provide evidence of drug interaction metformin, diflunisal, or either compound with thiazolidinediones lithium and carbon tetrachloride (at doses similar to those used for these trials).

5.6.9. Acesulfame-K – In vitro and vivo

In vitro and vivo animal testing has shown a lack of drug interaction metformin, diflunisal, or either compound with thiazolidinediones lithium and carbon tetrachloride (at doses similar to those used for these trials).

5.6.10. Atorvastatin – In vitro and vivo

With respect to the mechanisms of action atorvastatin in animals, vitro and vivo animal testing studies have failed to provide evidence of drug interactions with metformin, cholestyramine, or any of these compounds with lithium and carbon tetrachloride (at doses similar to those used for these trials).

In view of the present invention, certain embodiments, as disclosed herein, may involve a combination of glucagon-lowering agent like metformin with an insulin-lowering agent like dexamethasone, a C-peptide human insulin or its ester, an insulin-like growth factor (IGF) like human insulin, ghrelin, or its ester, a thiazolidinediones lithium and carbon tetrachloride, or a thiazolidinated nucleoside analog. further embodiment of the present invention includes a combination of C-peptide or an insulin like C2, C3, a ghrelin GHRL or its ester, an insulin like insulin, all of which may be used concurrently with the insulin-lowering agent.

5.6.11. Glulisine – In vitro and vivo

With respect to the mechanisms of action glulisine, in vitro and vivo animal testing has revealed a lack of drug interaction with metformin, cholestyramine, or any of these compounds with lithium and carbon tetrachloride (at doses similar to those used for these trials) or the thiazolidinediones lithium and carbon tetrachloride. In contrast with most of the other drugs this group, glulisine also has been shown to induce a biphasic antiapoptotic effect in the liver which may not be caused by lowering of cellular ATP levels and as so does not cause a paradoxical slowing down of the growth as found with glipizide.

6. Conclusions

There will be easy to follow graphical display of a list ingredients and their respective concentrations for each listed compound which are to be added a subject for the above purposes. These proportions of ingredients needed will depend on whether the subject wants to be given a single source drug or several drugs and on the particular form of subjects insulin-like growth factor (IGF) or the.

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References

1. Koo J, Choi HJK, Yoon G. Serum concentration of glucose-dependent insulinotropic polypeptide in patients with polycystic ovary syndrome: a multicenter, randomized, double-blind clinical trial. Clin Endocrinol (Oxf). 2004;59:2-9.

2. Hegde U, Vazquez O, Perez V, Pardo S, Jairou P, Lecache C, Soto-Gonzalez J. Polycystic ovary syndrome and insulin resistance in a Hispanic population. J Clin Endocrinol Metab. 2002;87:2549-2552.

3. Sattar N, Vazquez O, Jairou P, Lopez J, Gonzalez Moreno E. A multicenter study on the Atorva 2mg $50.75 – $0.42 Per pill relationship of insulin sensitivity and dosage in adolescents with polycystic ovary syndrome. Clin Endocrinol (Oxf). 2004;59():6-10.

4. Ezzati M, Jairou atorvastatin 40 mg kaufen P, Garcia O, Pardo S, Sattar N, Sanchez-Molina V. Effect of levothyroxine and deschloroketones on insulin sensitivity, blood pressure, and lipid profile in polycystic ovary syndrome. J Endocrinol Invest. 2004;29(Suppl 4):1041-1046.

5. Jairou P, Vazquez O, Sattar N, Jairou P. Effect of levothyroxine treatment on lipids, glucose, and insulin levels in polycystic ovary syndrome. J Clin Endocrinol Metab. 2004;87:2783-2789.

6. Fonseca M, Lecache C, Vazquez O, Jairou P. Lifestyle intervention in a polycystic ovary-attached population. Endocr Pract. 2004;5(Suppl 1):35-47.

7. Hernández-Chaverri S, Moreno P, Fonseca M. Impact of lifestyle modification on metabolic parameters in a polycystic ovary-attached population. Nutr Res Rev. 2004;20:63-74; discussion 83-85.

8. Yoon G, Koo J. Effect of high dose levothyroxine on metabolic outcomes and parameters in ovariectomized patients with polycystic ovary syndrome. Hum Reprod. 2005;21:2853-2857.

9. Vazquez O, Rovira J, Lopez Jairou P, Lecache C. Luteinizing hormone, insulin, glucose and lipid profiles in patients with polycystic ovary syndrome who discontinued levothyroxine treatment. Clin Endocrinol (Oxf). 2005;58:27-33.

10. Hernández-Chaverri S, Ezzati M, Fonseca M. Lifestyle intervention in women with polycystic ovary syndrome. Endocr Pract. 2005;6:1-15.

11. Ezzati M, Moreno P, Rovira J, Lopez D, Vazquez O, Jairou P. Diet, lifestyle and insulin parameters in a polycystic ovary-attached population. J Endocrinol Invest. 2005;29:941-948.

12. Gualano R, Lecache C, Gómez-Morillas J, Vazquez O, Jairou P. Diet and insulin resistance in women with polycystic ovary syndrome in a Mediterranean country. J Clin Endocrinol Metab. 2002;87:3851-3858.

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